Bcl-2 CLIA Kit
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- Target See all Bcl-2 (BCL2) CLIA Kits
- Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))
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Reactivity
- Mouse
- Detection Method
- Chemiluminescent
- Method Type
- Sandwich ELISA
- Detection Range
- 78 pg/mL - 5000 pg/mL
- Minimum Detection Limit
- 78 pg/mL
- Application
- ELISA
- Purpose
- Mouse B-Cell Leukemia/Lymphoma 2 (Bcl2) CLIA Kit is a Sandwich CLIA Kit for use with Serum, plasma, tissue homogenates and other biological fluids.
- Sample Type
- Plasma, Serum, Tissue Homogenate
- Analytical Method
- Quantitative
- Sensitivity
- < 30 pg/mL
- Featured
- Discover our best selling BCL2 ELISA Kit
- Top Product
- Discover our top product BCL2 ELISA Kit
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- Application Notes
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Stability: The stability of the kit is determined by the rate of activity loss. The loss rate is less than 5 % within the expiration date under appropriate storage conditions. To minimize performance fluctuations, operation procedures and lab conditions should be strictly controlled. It is also strongly suggested that the whole assay is performed by the same user throughout.
Recommended dilutions: Optimal dilutions/concentrations should be determined by the end user.
Standard Form: Lyophilized
- Plate
- Pre-coated
- Restrictions
- For Research Use only
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- Storage
- 4 °C/-20 °C
- Storage Comment
- Upon receipt, store the kit according to the storage instruction in the kit's manual.
- Expiry Date
- 6 months
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- Target See all Bcl-2 (BCL2) CLIA Kits
- Bcl-2 (BCL2) (B-Cell CLL/lymphoma 2 (BCL2))
- Alternative Name
- BCL2 (BCL2 Products)
- Synonyms
- Bcl-2 CLIA Kit, PPP1R50 CLIA Kit, bcl-2 CLIA Kit, AW986256 CLIA Kit, C430015F12Rik CLIA Kit, D630044D05Rik CLIA Kit, D830018M01Rik CLIA Kit, BCL-2 CLIA Kit, Bcl2 CLIA Kit, BCL2, apoptosis regulator CLIA Kit, B cell leukemia/lymphoma 2 CLIA Kit, B-cell CLL/lymphoma 2a CLIA Kit, B-cell CLL/lymphoma 2 protein CLIA Kit, BCL2 CLIA Kit, Bcl2 CLIA Kit, bcl2a CLIA Kit, BCL-2 CLIA Kit
- Pathways
- MAPK Signaling, PI3K-Akt Signaling, Apoptosis, Caspase Cascade in Apoptosis, Regulation of Muscle Cell Differentiation, Cell-Cell Junction Organization, Skeletal Muscle Fiber Development, Autophagy, Smooth Muscle Cell Migration, Negative Regulation of intrinsic apoptotic Signaling
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