Phone:
+1 877 302 8632
Fax:
+1 888 205 9894 (Toll-free)
E-Mail:
orders@antibodies-online.com

Prostate Specific Antigen ELISA Kit

PSA Reactivity: Human Colorimetric Sandwich ELISA
Catalog No. ABIN577067
  • Target See all Prostate Specific Antigen (PSA) ELISA Kits
    Prostate Specific Antigen (PSA)
    Reactivity
    • 14
    • 9
    • 7
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    Human
    Detection Method
    Colorimetric
    Method Type
    Sandwich ELISA
    Application
    ELISA
    Purpose
    This PSA enzyme linked immunosorbent assay (ELISA) applies a technique called a quantitative sandwich immunoassay. The microtiter plate provided in this kit has been pre-coated with a monoclonal antibody specific for PSA. Standards or samples are then added to the microtiter plate wells and PSA, if present, will bind to the antibody pre-coated on the wells. In order to quantify the amount of PSA present in the sample, a standardized preparation of horseradish peroxidase (HRP)-conjugated monoclonal antibody, specific for PSA are added to each well to
    Analytical Method
    Quantitative
    Sensitivity
    Estimated to be 1 ng/mL PSA antigen in human serum.
    Components
    Standards: 1 set/2 vials
    Top Product
    Discover our top product PSA ELISA Kit
  • Plate
    Pre-coated
    Restrictions
    For Research Use only
  • Preservative
    Without preservative
  • Petiwala, Berhe, Li, Puthenveetil, Rahman, Nonn, Johnson: "Rosemary (Rosmarinus officinalis) extract modulates CHOP/GADD153 to promote androgen receptor degradation and decreases xenograft tumor growth." in: PLoS ONE, Vol. 9, Issue 3, pp. e89772, (2014) (PubMed).

  • Target See all Prostate Specific Antigen (PSA) ELISA Kits
    Prostate Specific Antigen (PSA)
    Abstract
    PSA Products
    Background
    Hepatitis resulting from infection with viruses other than Hepatitis A Virus (HAV) and Hepatitis B (HBV) virus was previously referred to as non-A, non-B hepatitis. The first characterised non-A, non-B hepatitis agent was that responsible for parentally transmitted non-A, non-B hepatitis, or what is now called Hepatitis C Virus. This was followed by the cloning of a portion of the fecal-orally-transmitted agent, the Hepatitis E Virus (HEV). Hepatitis E Virus has been referred to as enterically transmitted non-A, non-B hepatitis. Epidemics of enterically transmitted Hepatitis E Virus have been recognised worldwide but occur principally in developing countries. They have been reported in Southeast Asia, central Asia, Africa, Mexico, and Central America. In these areas, contaminated water has been implicated as the principal vehicle of virus transmission. Although HEV and HAV are transmitted in a similar manner, there are major differences in the clinical, pathological, and epidemiological courses of these two viruses. In particular, the mortality rate for HEV infection is 1 to 2%, or approximately 1-fold greater than that seen for HAV. Infection with HEV is particularly fatal for pregnant women, for whom the mortality rate can be as high as 1 to 2%
    Pathways
    Complement System
You are here:
Support