HSV1 ICP0 Antibodies
(Herpes Simplex Virus Type 1 ICP0 (HSV1 ICP0))
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Recommended HSV1 ICP0 Antibodies
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Latest Publications for our HSV1 ICP0 products
: "Intrinsic innate immunity fails to control herpes simplex and vesicular stomatitis virus replication in sensory neurons and fibroblasts." in: Journal of virology, (2014) (PubMed).: "Herpes simplex virus requires poly(ADP-ribose) polymerase activity for efficient replication and induces extracellular signal-related kinase-dependent phosphorylation and ICP0-dependent nuclear ..." in: Journal of virology, Vol. 86, Issue 1, pp. 492-503, (2012) (PubMed).
: "Imiquimod suppresses propagation of herpes simplex virus 1 by upregulation of cystatin A via the adenosine receptor A1 pathway." in: Journal of virology, Vol. 86, Issue 19, pp. 10338-46, (2012) (PubMed).
: "HSV ICP0 recruits USP7 to modulate TLR-mediated innate response." in: Blood, Vol. 113, Issue 14, pp. 3264-75, (2009) (PubMed).
: "Multiple peptides homologous to herpes simplex virus type 1 glycoprotein B inhibit viral infection." in: Antimicrobial agents and chemotherapy, Vol. 53, Issue 3, pp. 987-96, (2009) (PubMed).
: "The cellular RNA export receptor TAP/NXF1 is required for ICP27-mediated export of herpes simplex virus 1 RNA, but the TREX complex adaptor protein Aly/REF appears to be dispensable." in: Journal of virology, Vol. 83, Issue 13, pp. 6335-46, (2009) (PubMed).
: "Transient expression of herpes simplex virus type 1 ICP22 represses viral promoter activity and complements the replication of an ICP22 null virus." in: Journal of virology, Vol. 83, Issue 17, pp. 8733-43, (2009) (PubMed).
: "Two small RNAs encoded within the first 1.5 kilobases of the herpes simplex virus type 1 latency-associated transcript can inhibit productive infection and cooperate to inhibit apoptosis." in: Journal of virology, Vol. 83, Issue 18, pp. 9131-9, (2009) (PubMed).
: "Hsc70 focus formation at the periphery of HSV-1 transcription sites requires ICP27." in: PLoS ONE, Vol. 3, Issue 1, pp. e1491, (2008) (PubMed).
: "Regulation of human endogenous retrovirus W protein expression by herpes simplex virus type 1: implications for multiple sclerosis." in: Journal of neurovirology, Vol. 12, Issue 1, pp. 65-71, (2006) (PubMed).