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GJB2 antibody

GJB2 Reactivity: Human, Rat ELISA, IHC Host: Rabbit Polyclonal unconjugated
Catalog No. ABIN7238093
  • Target See all GJB2 Antibodies
    GJB2 (Gap Junction Protein, beta 2, 26kDa (GJB2))
    Reactivity
    • 40
    • 23
    • 20
    • 5
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    Human, Rat
    Host
    • 42
    • 4
    • 1
    Rabbit
    Clonality
    • 43
    • 4
    Polyclonal
    Conjugate
    • 28
    • 3
    • 2
    • 2
    • 2
    • 2
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    • 1
    This GJB2 antibody is un-conjugated
    Application
    • 33
    • 25
    • 17
    • 6
    • 4
    • 3
    • 2
    • 1
    • 1
    • 1
    • 1
    ELISA, Immunohistochemistry (IHC)
    Characteristics
    Polyclonal Antibody
    Purification
    Affinity purification
    Immunogen
    Synthetic peptide of human GJB2
    Isotype
    IgG
    Top Product
    Discover our top product GJB2 Primary Antibody
  • Application Notes
    IHC 1:100-1:300
    Restrictions
    For Research Use only
  • Format
    Liquid
    Concentration
    2.3 mg/mL
    Buffer
    PBS with 0.05 % sodium azide and 50 % glycerol, PH7.4
    Preservative
    Sodium azide
    Precaution of Use
    This product contains Sodium azide: a POISONOUS AND HAZARDOUS SUBSTANCE which should be handled by trained staff only.
    Storage
    -20 °C
    Storage Comment
    Store at -20°C. Avoid freeze / thaw cycles.
  • Target
    GJB2 (Gap Junction Protein, beta 2, 26kDa (GJB2))
    Alternative Name
    Connexin-26 (GJB2 Products)
    Background
    This gene encodes a member of the gap junction protein family. The gap junctions were first characterized by electron microscopy as regionally specialized structures on plasma membranes of contacting adherent cells. These structures were shown to consist of cell-to-cell channels that facilitate the transfer of ions and small molecules between cells. The gap junction proteins, also known as connexins, purified from fractions of enriched gap junctions from different tissues differ. According to sequence similarities at the nucleotide and amino acid levels, the gap junction proteins are divided into two categories, alpha and beta. Mutations in this gene are responsible for as much as 50 % of pre-lingual, recessive deafness.
    NCBI Accession
    NP_003995
    UniProt
    P29033
    Pathways
    Sensory Perception of Sound, Cell-Cell Junction Organization
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